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discussion 263-4, 1995

These findings stress the importance of targeting mBzR/IMAC as a harbinger of more severe and permanent mitochondrial damage, which, through the ROS-induced ROS release mechanism, primes the opening of the PTP

Clinically, Se deficiency correlates with mitochondrial dysfunction and pathological accumulation of ROS/mtROS in chondrocytes, which contributes to OA progression, as evidenced by significantly lower serum Se levels in OA patients compared to healthy controls [20]

No more "muffin top." Now, despite menopause and the pandemic, Rebecca continues to feel healthy and strong with her hobby of renovating homes

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