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Because of this critical connection, it is not surprising that iatrogenic depletion of specific amino acids (i.e., methionine by methioninase 218 , arginine by arginase 219 ) is presently in clinical trials as tumor-selective therapeutic agents, reviewed in ref

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Hence, mutation of either C168 or C190 results in the loss of the redox responsiveness of GSAAT1 in planta

"A human vitamin B12 trafficking protein uses glutathione transferase activity for processing alkylcobalamins"

What they did: n = ~ 7,000 adults with obesity and high CV risk