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The study supports the concept that GHSs may provide a safer and more physiologic alternative to exogenous GH therapy by stimulating endogenous pulsatile GH release, thereby avoiding supraphysiologic IGF-1 levels [2]

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Pickart proposed that this activity in human plasma albumin was a tripeptide glycyl-L-histidyl-L-lysine and that it might function by chelating metal ions

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