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Fixed modification was set to carbamidomethylation of cysteines, and variable modifications were set to oxidation of methionines, asparagine and glutamine deamidation, and protein N-terminal acetylation

10.1186/s12935-022-02679-8 141 MarketM.TennakoonG.ScaffidiM.CookD

Mechanisms of disease: mitochondrial dysfunction in sensory neuropathy and other complications in diabetes

Moutailler S, Moro CV, Vaumourin E, Michelet L, Tran FH, Devillers E, Cosson JF, Gasqui P, Mavingui P, Vourch G, Vayssier-Taussat M

The findings of the study revealed that hyperoxia raised the initial amount of Nrf2 and many transcriptional targets including Immunoproteasome, 20S Proteasome, NQO1, Lon protease, and HO-1, but caused the loss of cellular capacity to adapt to signaling levels of H 2 O 2