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Notably, trophoblast cells may respond to maternal immune dysregulation by upregulating Fas ligand (FASL) and tumor necrosis factor receptors (TNFRs), thereby triggering apoptotic signaling and promoting trophoblast dysfunction (51)

We assume that at least two Cys residues of GSSAT are oxidized, which may form an intracellular disulfide bond under oxidizing conditions

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Prevalence of atopic comorbidities in eosinophilic esophagitis: A case-control study of 449 patients