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Notably, nanoparticle-based formulations of quercetin have been shown to suppress pro-inflammatory cytokine expression and enhance antioxidant defences through increased superoxide dismutase (SOD) activity, ultimately reducing cancer cell viability (Hannan et al

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Sulforaphane also induced S-phase and G2/M-phase cell cycle arrest, enhanced histone acetylation, and up-regulated cell cycle proteins in the prostate cancer cell lines, DU145 and PC-3 (Rutz et al., 2020), suggesting that sulforaphane treatment leads to reduced cell proliferation activity