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doi: 10.3390/ph16020271 100

The fact that it showed significant neuroprotection when administered just 30 seconds after ischemia suggests a rapid mechanism of action, which is critical in the narrow therapeutic windows that characterize acute brain injury

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Experimental work has confirmed its capacity to restore myotendinous junctions, promote tendon-to-bone repair, and support the recovery of severe musculoskeletal injuries that would not normally regenerate on their own

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