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This exacerbates lipid peroxidation, drives excessive ROS accumulation and neuroinflammation, and ultimately triggers ferroptosis, resulting in irreversible neuronal damage and neurotoxicity (Table 1), suggesting that therapeutic targeting of ferroptosis may offer a promising strategy for the prevention and treatment of NDDs

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Additionally, development of novel NAD+ precursors and delivery methods aims to improve bioavailability and tissue-specific targeting