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9 Figure 7 Hypothetical schemes for alcohol-mediated alveolar macrophage dysfunction

When these complexes are saturated, excess free Fe 2+ accumulates in the labile iron pool (LIP), accelerating fenton reactions and increasing OH and L-O levels, thereby promoting lipid peroxidation and inducing ferroptosis

doi:10.1016/s0165-0173(97)00045-3

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The compound is produced through controlled synthesis and metal-complexation processes and is provided exclusively for in vitro research in qualified laboratory settings