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One particularly instructive example comes from research using 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced models of PD, where chronic administration of monoacylglycerol lipase inhibitors was found to not only preserve dopaminergic neurons in the substantia nigra but also induce a distinctive microglial activation profile characterized by increased release of neurotrophic factors and decreased expression of classical inflammatory markers [459]

Identity is confirmed by matching the measured mass to the theoretical molecular weight of 1419.55 g/mol, which also flags any truncation in synthesis

Handa, Setsuko et al

Butner LE, Fulco PP, Feldman G, et al

Failure to promptly repair these damages may lead to genomic instability and abnormal cellular function, and may even drive pathological processes such as cancer, neurodegenerative diseases and autoimmune diseases (1,4-6)