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Vergauwen, B., Elegheert, J., Dansercoer, A., Devreese, B

These include mutations in genes such as: SNCA Alpha-synuclein gene PRKN Parkin RBR E3 Ubiquitin Protein Ligase PINK1 Gene PARK6 DJ-1 and PARK7 Gene LRRK2 It is believed that mutations in these genes can cause dysfunction and eventual death of dopamine-producing neurons through various mechanisms, including protein aggregation, mitochondrial dysfunction, and oxidative stress

Moreover, some mRNA and viral vector vaccines can induce Th1 cell responses 19,20 and persistent human germinal center responses, 21,22 which provide more efficient protection

Patients with more severe disease have higher levels of S2-, S1-, and RBD-binding antibodies and concomitantly the strongest serum nAb titers at two to four weeks after infection [85]