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In support of this assumption are findings of haloperidol-induced cytotoxicity in human ACs [179, 180], reduction of AC (but not OL) densities in macaque monkey brains after chronic exposure to antipsychotics [97], and effects of chronic antipsychotic medication on AC marker expression in rat brains [181]

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A similar interaction in GSH metabolism by cysteine supply via GSH export appears to take place between astrocytes and endothelial cells, and this cysteine shuttle has been reported to help to maintain endothelial barrier stability and integrity (Huang et al

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