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The other argument is that gout is primarily an inflammatory disease resulting in systemic increases in proinflammatory factors 20

In the gut, T cells maintain immune balance, differentiating into proinflammatory (Th1, Th17) or anti-inflammatory [Th2, regulatory T cells (Tregs)] subsets (Dressman and Elyaman, 2022)

Consequently, mitochondrial metabolism and signaling including the UPR mt have been implicated in integrating nutrient- and microbiota-derived signals in the intestinal stem cell niche 8 , critically affecting the regenerative capacity of the epithelium under homeostatic and disease conditions

Novel ferroptosis inhibitors with improved potency and ADME properties

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