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Dysbiosis leads to altered levels of key metabolites: reduced SCFAs (e.g., butyrate, acetate) impair support for Treg cell differentiation and inhibition of Th17 responses, while compromising intestinal barrier integrity

This review highlights oxidative stress as a major contributor to brain aging and neurological disease, with the brain particularly vulnerable due to its high metabolic activity

While its primary action involves p53 release, the modulation of FOXO4-related pathways intersects with cellular stress-response systems

Relative expression levels were quantified using ImageJ software (National Institutes of Health)

doi: 10.1017/S0007114510002400 111 HananiM