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Additionally, glutathione injection is essential for restoring mitochondrial function, which is crucial for efficient cellular signaling and metabolic processes

Therefore, a better understanding of the dysregulation of the antioxidant KEAP1-NRF2 activity in ALS could help to develop novel therapeutic strategies able to halt disease progression, prolonging patients survival

Goel P, Chakrabarti S, Goel K, Bhutani K, Chopra T, Bali S (2022) Neuronal cell death mechanisms in Alzheimers disease: an insight

FLT3 Inhibitor Treatment in FLT3-Mutated AML Is Associated with Development of Secondary FLT3-TKD Mutations

NF-B is also involved in this process (Yang et al., 2012a)