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Atovaquone, a repurposed antimalarial drug, acts as a selective OXPHOS inhibitor by targeting the CoQ10-dependent mitochondrial complex III, causing electron leakage, ROS production, and cell death, as observed in Fhit-deficient lung cancer cell lines

OU749 is an uncompetitive inhibitor with a K i of 17 uM [76], while GGsTop is a mechanism-based irreversible inhibitor, with an active ester of phosphonate mimicking the transition state of GSH in the GGT pocket, whose k inact /K I is 57 M 1 s 1 [77]

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Reactive oxygen species (ROS) or free radicals cause damage to key cellular components like DNA, RNA, lipids, proteins, and structural membranes

In the cellular context, these aberrant interactions can disrupt physiological processes by sequestering various proteins essential for normal cellular functions, resulting in diffused aggregates that may act as molecular triggers for LB nucleation