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Despite decades of research, the etiology and pathophysiological mechanisms underlying SCZ remain poorly understood

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689 Clinical analyses of differences in the expression of ferroptosis-related genes in blood samples from SCI patients and healthy controls confirmed the upregulation of TLR4, STAT3, and HO-1, consistent with the bioinformatics results

Indeed, GSH, Trx, Grx, Prx (together with the enzymes catalyzing their reduction), and the enzymatic antioxidant defense, as a whole, concur to modulate either ROS concentration, or the availability of cellular thiols, thus regulating the function of a large number of proteins implicated in the induction and/or execution of redox-sensitive pathways

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