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GSH can modulate the activity of various redox-sensitive signaling pathways, including the nuclear factor erythroid 2-related factor 2 (Nrf2) pathway, which regulates the expression of antioxidant and detoxification genes, and the mitogen-activated protein kinase (MAPK) pathway, which regulates cell proliferation, differentiation, and apoptosis

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AMPK activation of muscle autophagy prevents fasting-induced hypoglycemia and myopathy during aging

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The presence of CEAID did not affect the response to treatment or the long-term outcomes in a large study including adult patients with AIH [39]