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However, the recovery of the cell growth by overexpression of SHM2 and/or CYS4 in GCI/ SERs seemed to be the result that surplus biosynthesized l-Ser was metabolizing to Gly and l-Cys (Fig

b , Cell death quantification (% trypan blue + ) of glucose-deprived shCtrl and shGDH1 Huh7 and H1299 cells treated with dimethyl succinate (+,1 mM, ++, 5 mM)

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