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There is concern that high levels of added antioxidants from concentrated supplements may help protect the cancer cells from radiation or chemotherapy (which often work by creating oxidative damage)

By understanding the metabolic pathway disruption and other pathophysiological determinants that dismantle ferroptosis defence, this may expose druggable targets to restore metabolic flux and recover ferroptosis defences in disease contexts

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Every structural type of GS presents a composite active site where catalytic residues are contributed from two different protomers of the complex (Figure 1)

BMC Genomics 15:753