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As a consequence, the proton gradient generated by the ETC is consumed as the ATP is produced and has to be constantly re-established using reduced coenzymes, NADH, and FADH 2 (Figure 1)

[DOI] [PMC free article] [PubMed] [Google Scholar] 86.Bloom H.L., Shukrullah I., Veledar E., Gutmann R., London B., Dudley S.C

In apoptosis, -sitosterol upregulates pro-apoptotic factors such as p53 and Bax, while downregulating anti-apoptotic proteins such as Bcl-2, leading to an increase in mitochondrial outer membrane permeability and the activation of key apoptotic enzyme cascades such as caspase-9 and 3, which induces programmed cell death (83)

Neuroinflammation 215 LeeM.KimC.ShinS.MoonS.ChunM

doi: 10.1016/j.cell.2015.12.034, PMID: 29 AragonsGRowan SGFranciscoSYangWWeinbergJTaylorAet al