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On the pharmacology side, HGF-induced MET activation exerts beneficial neuroprotective effects also in adulthood, specifically in neurodegenerative disease, and in preclinical models of cerebral ischemia, spinal cord injuries, and neurological pathologies, such as Alzheimers disease (AD), amyotrophic lateral sclerosis (ALS), and multiple sclerosis (MS)

Nevertheless, all tested potential NUPR1-regulated targets were modulated as predicted by IPA in our culture system except for CEP170B, which confirms them as NUPR1-dependent targets also in human bronchial epithelial cells

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modulates immune aging and protects pancreatic islet cells in autoimmune diabetes models [5] [10]

Since the approval of semaglutide as Wegovy and tirzepatide as Zepbound, pharmaceutical companies and investors have been watching closely for the next therapy capable of matching or exceeding their clinical benchmarks