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The high energy demand of the brain, the gate-keeping function of the bloodbrain barrier (BBB), a high lipid content, vulnerable neuronal subpopulations, and glutamatergic neurotransmission all make the brain particularly vulnerable against mitochondrial dysfunction, neuroinflammation and excitotoxicity (Klker et al

Colon-Soto, M., Peredo, R

Non-alcoholic fatty liver disease and the metabolic syndrome: An update

Others found decreased Mfn1, Mfn2, and Opa1 levels combined with increased mitochondrial fission in primary neurons from Fmr1 -KO mice [334]

The differentially expressed lncRNA-46546 between Rose Crown and Cobb broiler embryos has been discovered to promote TG synthesis and lipid droplet accumulation in intramuscular preadipocytes through cis-regulating its target gene, 1-acylglycerol-3-phosphate-O-acyltransferase 2 ( AGPAT2 ) [98]