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NAC appears to exert its biological activity by acting as a precursor of glutathione, a ubiquitous compound with anti-oxidant properties that is involved in the metabolism of xenobiotics, signal transduction and gene expression, regulation of the cell cycle and apoptosis, and prostaglandin and leukotriene synthesis, among other processes [1, 2]

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Accordingly, agents that restore glutathione-glutamate homeostasis may afford therapeutic benefit