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Key mechanisms of action include: Fibroblast activation Suppression of collagen-degrading enzymes Reduced inflammatory cytokines Enhanced granulation tissue formation Improved angiogenesis In ischemic wound models, injectable GHK-Cu restored fibroblast function and significantly accelerated skin repair compared to controls

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Besides the cytotoxicity pathways discussed above, Se-induced cytotoxicity against malignant cells might also be the result of ROS generation, as in the case of injection of biogenic SeNPs into the abdominal cavity of mice following their inoculation with highly malignant H22 hepatocarcinoma cells [294]

Biomedicines 10:2236

Uses & Potential Benefits Here are the commonly cited uses and what the research actually shows