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They likely reflect the difference between pathological IGF-1 excess (which does appear to increase cancer risk and accelerate ageing in some models) and normalisation of IGF-1 in individuals whose levels have declined below an optimal physiological range due to somatopause

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Both agents continued to induce -SMA expression, collagen transcription, and ERK1/2 activation, with similar dynamics as in wild-type cells

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Yet in contrast to lack of data, the promotion is boundless economist.com/science-and-te More in depth here erictopol.substack.com/p/the-peptide- The email I received today to promote peptides The answer isnt no, even based on your stated standards The answer is TBD more data is needed to confirm or deny the anecdotal reports Join the conversation