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In contrast, an arrestin is involved in hDOR internalization but not desensitization upon DPDPE (Aguila et al., 2003b) while upon DPDPE exposure, the receptor is mainly targeted to lysosomes for degradation (Marie et al., 2003b) probably by a mechanism involving GASP (Whistler et al., 2002)

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+ Magnesium Chelate Blend (Gluconate, Acetyl Taurinate and Citrate) specifically chosen due to their high bioavailability and easy absorption into the gut with minimal discomfort*

(July 1995)

Stability in the stomach does not automatically confer meaningful systemic absorption it is a necessary but not sufficient condition for oral efficacy