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R., Chen, L., Neuhofer, P., Garbuzov, A., et al

However, genome-wide association study (GWAS) 168,169,170 have revealed a host of relatively novel risk loci and their genes, with limited but emerging evidence that some could be implicated in both mitophagy and ROS, including VPS13C 171 and SREBF1 172

Treating cells with L-buthionine sulfoximine (BSO), an inhibitor that blocks the synthesis of GSH, renders neuronal SH-SY5Y cells expressing mutant valosin-containing protein (VCP) R487H more susceptible to oxidative stress (Hirano et al., 2015) and increases mutant SOD1 toxicity (Alvarez-Zaldiernas et al., Drosophila models increases markers of oxidative stress, including protein carbonylation and glutathione S transferase D1 (Duan et al., 2010

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Absence of these enzymes would therefore result in accumulation of homocysteine in the parasite