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Excessive ROS not only directly causes oxidative damage to retinal cells and initiates retinal neovascularization, but also impairs the structural and functional integrity of the BRB

Antioxidant enzyme activities and oxidative stress in affective disorders

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In addition, T cells exhibited exhaustion phenotypes with the expression of higher levels of exhaustion markers including programmed cell death protein-1 (PD-1) and T cell immunoglobulin and mucin domain-3, suggesting that these T cells may have regulatory activities

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