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6.1.3 Complex I hypofunction stratification Multi-cohort and patient-derived data show that a biologically meaningful subset of idiopathic PD exhibits mitochondrial complex I deficiency aligned with higher ROS burden and clinical heterogeneity, supporting mitochondria-linked oxidative stress as a disease-relevantnot merely genericaxis (127)

doi: 10.1038/s41419-021-03813-6 149 HuangJKuangWZhouZ

Other Names for This Condition Hyperammonemia, type III N-acetylglutamate synthetase deficiency NAGS deficiency Additional Information & Resources Genetic Testing Information Genetic and Rare Diseases Information Center Patient Support and Advocacy Resources Clinical Trials Catalog of Genes and Diseases from OMIM Scientific Articles on PubMed References Caldovic L, Morizono H, Panglao MG, Cheng SF, Packman S, Tuchman M

61,62 The acetylation of p53 at certain sites hinders the phosphorylation of several serines located in the p53 NTD, which activates genes with high p53 affinity, including CDKN1A

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