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Beyond current applications in myocardial infarction repair, cardiac fibrosis reduction, and angiogenesis promotion, future investigations of HA in cardiovascular diseases should explore its role in modulating immuneinflammatory responses that drive vascular remodeling and heart failure progression, particularly through targeted regulation of macrophage and fibroblast phenotypes

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Strikingly, sporadic mutation of equivalent arginine residues in the HRD motifs of unrelated protein kinases are similarly catastrophic and cause rare, monogenic forms of various human diseases [145,146,147,148,149]

In: Difficult Decisions in Thoracic Surgery: An Evidence-Based Approach