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Mutagenesis experiments conducted on clathrin binding sites revealed a consistent preference of Ent1 for the Clathrin box, with the Arrestin and W-boxes appearing to play a less critical role for Ent1, serving as secondary sites of interaction

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Recent studies have highlighted the involvement of tripartite motif-containing 29 (TRIM29) and PERK in this process

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Ido A, Numata M, Kodama M, Tsubouchi H

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