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loss of lobular plates, architecture, and/or occurrence of cellular atypia

Axonal damage, evaluated by the presence of amyloid precursor protein (APP + ) deposits, firstly appears at remission (Fig

Some studies have proposed the hypothesis that the increase of mannose level in the CSF of PD patients may be directly related to the decreased MBL level that leads to decreased mannose binding, thus increasing the accumulation of free mannose in the CSF

doi:10.1016/j.ejca.2010.11.003

This hypercatabolism is evident by our data demonstrating depletion of amino acids and upregulation of the urea cycle as judged by elevated levels of urea cycle metabolites such as citrulline