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We next performed a correlation analysis to assess the mechanism of gut microbiota in PSO, which showed that Escherichia-Shigella was positively correlated with serum levels of Total Bile Acids (TBA) and negatively with levels of Hemoglobin (Hb), Direct Bilirubin (DBIL), and Indirect Bilirubin (IBIL) in patients with PSO

This perspective is strengthened by recent findings demonstrating that lipid-rich structures, most notably the myelin sheaths that envelop axons, can act as significant oxygen reservoirs

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They can disrupt bacterial membranes, produce reactive oxygen species that damage cellular macromolecules, and induce apoptosis through oxidative stress trails [131]