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ACTL6A impacts GSH de novo synthesis mainly by upregulating -glutamyl-cysteine synthesis To further assess the mechanism by which ACTL6A regulates GSH metabolism, we investigated the metabolic fate of U- 13 C glucose by liquid chromatography with mass spectrometry (LCMS), which produces -GC containing two 13 C atoms via GSH de novo synthesis pathway, GSH containing two 13 C atoms via GSH de novo synthesis or the SGOC pathway, or four 13 C atoms via both pathways (Fig

Chiang S, Kalinowski DS, Dharmasivam M, Braidy N, Richardson DR, Huang MLH (2020) The potential of the novel NAD+ supplementing agent, SNH6, as a therapeutic strategy for the treatment of Friedreichs ataxia

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We questioned whether chronic stress may exacerbate phenotypes and found that chronic stress significantly resulted in increased phosphorylated, insoluble TDP-35 fragments which then recovered upon removal of stress

5-HMC in the brain is abundant in synaptic genes and shows differences at the exon-intron boundary