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They influence the initiation, progression, and resolution of neuroinflammation through multiple mechanisms, including the release of inflammatory mediators, regulation of immune cell infiltration, interactions with microglia and endothelial cells, inflammasome activation, extracellular matrix degradation, and phenotypic plasticity (Tastan and Heneka, 2024

Transfer the contents of Vial 1 to Vial 2

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There were no serious or life-threatening AEs and all events resolved by the end of the study

KYN can also produce kynurenic acid (KA), which under physiological conditions, is attributed to neuroprotective effects