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Therefore, we used an objective protocol to iteratively eliminate patients from the analysis to limit the recurrence of individual variants (see the Methods section and Additional file 7: Table S5)

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The complex interaction between hyperhomocysteinemia and PD induces different pathological changes, including DNA hypomethylation, neuroinflammation, oxidative stress, and neuronal cell deaths (Doherty 2013)

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[16] Further research is needed, but this supports the idea that toxic exposure, like [17] nickel toxicity , may play a role in the development of nodules in those with Hashimotos