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The sterol-binding activity of PATHOGENESIS-RELATED PROTEIN 1 reveals the mode of action of an antimicrobial protein

Efferocytosis of apoptotic neutrophils reprogrammed macrophage mitochondrial metabolism through mieloperoxidase-dependent UCP2, which increases proton conductance in the inner mitochondrial membrane, thereby impairing reactive oxygen species (mtROS) production in response to bacteria