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These findings will be revisited in the Therapeutic Strategies section, but mechanistically they underscore that DAMPsening the mtDAMPs-innate receptor axis is a viable approach to reduce surgical organ damage

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Continuous tumor antigen stimulation and immune activation prompt a state of exhaustion or remodeling in the immune effector cells in the TME, rendering these cells unable to perform their normal functions

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These, in turn, can alter the brain and behavior or cause damage