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The rationale is mechanistic: BPC-157 and TB-500 appear to target complementary aspects of tissue repair

The GHK-Cu complex depends on the copper(II) oxidation state to maintain its coordination geometry and the biological activity attributed to that complex

However, multiple limitations in preclinical studies reduce the significance of current findings

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However, preclinical successes were not successfully translated into early clinical trials (e.g., VNP20009) due to a critical immunogenicity-safety trade-off, in which the attenuation required to ensure safety compromised the ability to generate effective anti-tumor immunity