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Traditionally treatment of lcFAODs consists of prevention of catabolism, LCT restriction, and supplementation of MCT [143, 145, 146]

In this study, we established an obesity (induced by HFD)-related AF mice model, which showed increased AF vulnerability and exhibited apparent atrial structural remodeling (Figures 1, 2)

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[7] Cleveland Clinic

Another mechanism for the development of CRPC is ligand promiscuity, which results from AR gene mutations that cause amino acid substitutions in the LBD that decrease specificity and selectivity for ligands ( eg , T877A, L701H, W741L, and F876L)