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Finally, translational progress is bottlenecked by the lack of strategies to selectively manipulate p53s ferroptosis-regulating activity: activating its pro-ferroptotic function in cancer cells while preserving its anti-ferroptotic role in normal tissues remains a major challenge, and no validated biomarkers exist to predict which tumors will respond to p53-targeted ferroptosis therapies

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Abnormal stress signaling has been noted in patients with fibromyalgia and other chronic pain disorders

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On the other hand, at low levels, cadaverine is imported to the cytoplasm of macrophages by L-lysine transporters, inducing a modest increase in glycolysis that supports Oxphos and itaconate generation