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The Jackson Laboratory), overexpressing familial AD mutant forms of human APP and PS1 transgenes under the transcriptional control of neural-specific elements of the mouse Thy1 promoter 100 , were used with a paternal transgenic inheritance 101

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Inhibition of transforming growth factor beta-activated kinase 1, NF-B, and non-receptor tyrosine kinases, which are involved in MAVS activation, resulted in reduced Siglec1 expression and interferon-inducible protein-10 secretion in macrophages [41, 143]

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Table 1 Drugs for improving heart failure in DCM and alleviating cardiovascular and cerebrovascular complications