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Abstract Allosteric modulation of ligand-gated ion channels provides a powerful mechanism to fine-tune their activity without competing with endogenous ligands

To view a copy of this license, visit About this article Cite this article Maiuri, L., Raia, V

3.3 Mechanistic target of the rapamycin Particularly, when ACSL4 was overexpressed, mTOR phosphorylation greatly increased, and when ACSL4 was downregulated, mTOR phosphorylation significantly decreased