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Integrative proteomic and transcriptomic analysis identifies TrkB (NTRK2) as a therapeutic target and a potential driver of EGFR inhibitor resistance in lung squamous cell cancer

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Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and central nervous system inflammation via the aryl hydrocarbon receptor

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As expected, Stattic and TRi-1 converted TGR to a pro-oxidant enzyme, whereas our non-covalent inhibitors did not increase NADPH consumption or superoxide production (Fig

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