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In terms of iron metabolism and homeostasis in immune cells, a study revealed that iron overload promotes abnormal differentiation of pathogenic T cells, including follicular helper T cells, by regulating DNA demethylation, thus exacerbating autoantibody production and the development of systemic lupus erythematosus (SLE) [26]

They show up in substrate oxidation studies, nitrogen balance measurements, and body composition tracking

Acknowledgements This work was supported by the National Research Foundation of Korea (NRF) grant funded by the Korea government (MSIP) (No

CBS constructs were diluted in 25 mM HEPES, pH 7.5, 200 mM NaCl, and 2.0 mM TCEP

[DOI] [PMC free article] [PubMed] [Google Scholar] 39.Liu, L