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determined the preventive mechanisms of delphinidin on disused muscle atrophy using in vitro and in vivo experimental models and concluded that delphinidin may prevent muscle atrophy via upregulation of miR-23a and suppression of MuRF1 expression (Murata et al

In parallel, treatment of normal melanocytes with the mTORC1 inhibitor rapamycin down-modulated pS6, indicating the efficacy of its action (Fig

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Notably, while we observed a robust reduction in the number of erythroblasts at all differentiation stages, we did not detect a defect in other lineages (Extended Data Fig