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Activated cisplatin then crosslinks two purine bases (adenine/guanine) of DNA by reacting with nitrogens at position seven of the purine rings, thus interfering with cell division and transcription of messenger ribonucleic acid (mRNA) (Dasari and Tchounwou, 2014)

Further knockdown of ACSL4 significantly abolished sorafenib-induced ferroptotic cancer cell death and tumor growth inhibition

Ginsenosides reverse tumor drug resistance Ginsenoside Rg3 has been the subject of extensive investigations regarding its potential to counteract the multidrug resistance of cancer cells

10.1039/C9QM00732F

[DOI] [PubMed] [Google Scholar] 83.van Eunen K, Bouwman J, Daran-Lapujade P, Postmus J, Canelas AB, Mensonides FIC, Orij R, Tuzun I, van den Brink J, Smits GJ, van Gulik WM, Brul S, Heijnen JJ, de Winde JH, Teixeira de Mattos MJ, Kettner C, Nielsen J, Westerhoff HV, Bakker BM