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Oxidized Mitochondrial DNA Activates the NLRP3 Inflammasome during Apoptosis

Krzystyniak, A [29] and Qu, Y [19] respectively conducted studies on depression using chronic unpredictable stress (CUS) and knockout of Nrf2 as models

Consistent with this phenotype, our mutants accumulated and secreted high quantities of amino acids and other weak acids (e.g., lactic acid, dicarboxylic acids, keto acids), which strongly alludes to dysregulated cellular metabolism (Fig

The fact that ROS was generated explosively by GSTP-silencing and the incomplete recovery of mitochondrial function by NAC demonstrated in this study indicates that GSTP-silencing induces oxidative stress through mitochondrial dysfunction

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