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5.2 Suppression of pro-inflammatory signaling (NF-B and MAPKs) Several organosulfur compounds (OSCs), including allicin, ajoene, and diallyl disulfide (DADS), attenuate canonical pro-inflammatory signaling by targeting redox-sensitive components of upstream kinase pathways

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These results suggest that administration of the Si-based agent significantly alleviated UC inflammation symptoms via the inhibition of pro-inflammatory humoral factor expression

Reducing oxidative stress in the mitochondria and cell membranes decreases ischemic injury and end-organ damage to the retina

However, this response is enhanced with LPC treatment and is mitigated by 1 week post-treatment